Neurodegenerative Disorders and LNP/mRNA.
Theoretical, histological and clinical evidence of protein deposition disease related to GTPs (Gene Therapy products) is presented.
I. What are Neurodegenerative Disorders?
From the Cleveland Clinic:
https://my.clevelandclinic.org/health/diseases/24976-neurodegenerative-diseases
Protein deposition, protein misfolding (abnormal three dimensional structure), demyelination and direct neuronal injury have been implicated as causative agents in these diseases.
A related series of disorders are classified as amyloid disorders.
Amyloidosis is a group of rare diseases caused by the accumulation of clumps of misfolded proteins, called amyloid fibrils, in organs and tissues in the body. This can happen almost anywhere, from the heart to the brain to the liver to the skin, to name a few. The buildup of amyloid fibrils can change the structure of those organs and tissues, which, in turn, affects their ability to function normally. Without treatment, amyloidosis can result in organ failure and depending on the organs involved, it can be lethal. https://ym.care/5wg
These diseases typically develop over years.
Katsuno M, Sahashi K, Iguchi Y, Hashizume A. Preclinical progression of neurodegenerative diseases. Nagoya J Med Sci. 2018 Aug;80(3):289-298. doi: 10.18999/nagjms.80.3.289. PMID: 30214078; PMCID: PMC6125655.
A more aggressive form of disease cause by misfolded proteins is called Prion Disease.
II. Didn’t pre-clinical studies of BNT162b2 address production of harmful proteins translated from transfecting mRNA in experimental animals?
NO. None were done.
From Pfizer’s pre-clinical study it is clear this topic was not studied.
“The protein encoded by the RNA in BNT162b2 is expected to be proteolytically degraded like other endogenous proteins. RNA is degraded by cellular RNases and subjected to nucleic acid metabolism. Nucleotide metabolism occurs continuously within the cell, with the nucleoside being degraded to waste products and excreted or recycled for nucleotide synthesis. Therefore, no RNA or protein metabolism or excretion studies will be conducted.” P20¶3
This statement about protein is nonsensical as some proteins are resistant to human proteases (enzymes that degrade proteins). The second statement about mRNA is simply false. One of the nucleotides that occur naturally in humans was replaced by a synthetic nucleotide never used in humans and designed to persist an unknown period of time but lasts months in some individuals.
III. What is the evidence of such abnormal proteins and related disease resulting from LNP/mRNA?
a. Theoretical basis of protein deposition disease from LNP/mRNA.
Dr. Kevin McCairn has been speaking on this topic as well in the context of Cruetzfeld-Jacob, Parkinson, and Alzheimer Disease.
b. Histopathological Evidence of protein deposition following treatment with LNP/mRNA.
Dr. Burkhardt’s group found evidence of spike protein deposition surrounding the splenic artery in the tissues of a person who died after injection of LNP/mRNA.
More amyloid like deposits (arrows) after mRNA therapy.
From Burkhardt et al, “This is spleen and was found in the biopsy specimen of this lady that I showed you before (Case 39). So, she has some amyloid and, certainly, this has some meaning for the function of the vascular tissue. Also, we have these deposits in the brain.”
c. Clinical Evidence of neurological protein deposition disease provided by Dr. Makis, MD.
Dr. Makis concludes,
“There are prion genetic sequences in the COVID-19 vaccine mRNA spike protein genetic code. What are they doing there???
This means that there is a currently unknown level of risk, in some people to PRODUCE PRION PROTEINS following COVID-19 vaccination, which is 100% fatal.”
Well done Dr.
God bless you. God bless us all.
Fight on.